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On the other hand, COX-1 selective inhibitor, ketorolac, and the non-selective COX-inhibitor, indomethacin -but not the COX-2 inhibitor- partially reversed the NPW-induced alleviation in gastric oxidative injury, indicating that the antioxidant properties of NPW involve a COX-1-dependent mechanism

We propose that dysbiosis promotes ferroptosis in ovarian granulosa cells through disrupted iron homeostasis, diminished production of protective microbial metabolites, and systemic inflammation, collectively contributing to the reproductive and metabolic disturbances in PCOS

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